The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal
The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal
MOAG-4 promotes the aggregation of α-synuclein by competing with self-protective electrostatic interactions - Journal of Biological Chemistry
Identification of MOAG-4/SERF as a Regulator of Age-Related Proteotoxicity: Cell
Polyglutamine protein aggregates are dynamic | Nature Cell Biology
Bag1–Hsp70 mediates a physiological stress signalling pathway that regulates Raf-1/ERK and cell growth | Nature Cell Biology
Probiotic Bacillus subtilis Protects against α-Synuclein Aggregation in C. elegans - ScienceDirect
C. elegans maximum velocity correlates with healthspan and is maintained in worms with an insulin receptor mutation | Nature Communications
Modulation of in Vivo HSP70 Chaperone Activity by Hip and Bag-1* - Journal of Biological Chemistry
Frontiers | Regulation of Age-Related Protein Toxicity | Cell and Developmental Biology
Frontiers | Cellular Regulation of Amyloid Formation in Aging and Disease | Neuroscience
The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal
Protective role of DNJ-27/ERdj5 in Caenorhabditis elegans models of human neurodegenerative diseases. - Abstract - Europe PMC
Frontiers | In vivo Pooled Screening: A Scalable Tool to Study the Complexity of Aging and Age-Related Disease | Aging
Protective role of DNJ-27/ERdj5 in Caenorhabditis elegans models of human neurodegenerative diseases. - Abstract - Europe PMC
Molecular chaperones and Parkinson's disease - ScienceDirect
Serum tryptophan decrease correlates with immune activation and impaired quality of life in colorectal cancer | British Journal of Cancer
The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal
The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal
Θορυβώδης οπουδήποτε Έρνεστ Σάκλετον calvin klein punk ευσαρκία λιοντάρι ζω
Θορυβώδης οπουδήποτε Έρνεστ Σάκλετον calvin klein punk ευσαρκία λιοντάρι ζω
Protective role of DNJ-27/ERdj5 in Caenorhabditis elegans models of human neurodegenerative diseases. - Abstract - Europe PMC
Frontiers | Membrane Interactions and Toxicity by Misfolded Protein Oligomers | Cell and Developmental Biology
Neurodegenerative diseases: Lessons from genome‐wide screens in small model organisms | EMBO Molecular Medicine
